Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88
ARTIGO
Inglês
Agradecimentos: This work was supported by Fundação de Amparo a Pesquisa do Estado de São Paulo (FAPESP, grant numbers 11/15682-4, 12/02270-2, and 15/18121-4) and Conselho Nacional de Desenvolvimento Cientı´fico e Tecnológico (CNPq, Regenera INCT Process grant 465656/201
The underlying mechanism by which MyD88 regulates the development of obesity, metainflammation, and insulin resistance (IR) remains unknown. Global deletion of MyD88 in high-fat diet (HFD)fed mice resulted in increased weight gain, impaired glucose homeos
FUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULO - FAPESP
11/15682-4; 12/02270-2; 15/18121-4
CONSELHO NACIONAL DE DESENVOLVIMENTO CIENTÍFICO E TECNOLÓGICO - CNPQ
465656/2014-5
aberto
Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88
Dectin-1 activation exacerbates obesity and insulin resistance in the absence of MyD88
Fontes
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Cell reports Vol. 19, no. 11 (June, 2017), p. 2272-2288 |