Pancreatic alpha-cell dysfunction contributes to the disruption of glucose homeostasis and compensatory insulin hypersecretion in glucocorticoid-treated rats
Alex Rafacho, Luiz M. Gonçalves-Neto, Junia C. Santos-Silva, Paloma Alonso-Magdalena, Beatriz Merino, Sebastião R. Taboga, Everardo M. Carneiro, Antonio C. Boschero, Angel Nadal, Ivan Quesada
ARTIGO
Inglês
Glucocorticoid (GC)-based therapies can cause insulin resistance (IR), glucose intolerance, hyperglycemia and, occasionally, overt diabetes. Understanding the mechanisms behind these metabolic disorders could improve the management of glucose homeostasis
CONSELHO NACIONAL DE DESENVOLVIMENTO CIENTÍFICO E TECNOLÓGICO - CNPQ
471397/2011-3
FUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULO - FAPESP
Aberto
Pancreatic alpha-cell dysfunction contributes to the disruption of glucose homeostasis and compensatory insulin hypersecretion in glucocorticoid-treated rats
Alex Rafacho, Luiz M. Gonçalves-Neto, Junia C. Santos-Silva, Paloma Alonso-Magdalena, Beatriz Merino, Sebastião R. Taboga, Everardo M. Carneiro, Antonio C. Boschero, Angel Nadal, Ivan Quesada
Pancreatic alpha-cell dysfunction contributes to the disruption of glucose homeostasis and compensatory insulin hypersecretion in glucocorticoid-treated rats
Alex Rafacho, Luiz M. Gonçalves-Neto, Junia C. Santos-Silva, Paloma Alonso-Magdalena, Beatriz Merino, Sebastião R. Taboga, Everardo M. Carneiro, Antonio C. Boschero, Angel Nadal, Ivan Quesada
Fontes
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PLoS one Vol. 9, no. 4 (Apr., 2014), p. 1-11, n. art. e93531 |