The contribution of eimeria coinfection and intestinal inflammation to cecal colonization and systemic spread of salmonella typhimurium deficient in tetrathionate reductase or type III secretion systems salmonella pathogenicity island 1 or 2

The contribution of eimeria coinfection and intestinal inflammation to cecal colonization and systemic spread of salmonella typhimurium deficient in tetrathionate reductase or type III secretion systems salmonella pathogenicity island 1 or 2

Claire-Sophie Rimet, John J. Maurer, Roy D. Berghaus, Brian J. Jordan, Luciana Helena Antoniassi da Silva, Lisa J. Stabler, Kasey K. Johnson, Laura R. Tensa, Karen M. Segovia, Monique S. Franca

ARTIGO

Inglês

Intestinal inflammation may provide a growth advantage for Salmonella and enhance its systemic spread in chickens. Salmonella triggers intestinal inflammation in the host by using type III secretion systems (T3SS) and produces the inflammatory end product tetrathionate. In mice, tetrathionate...

Fechado

The contribution of eimeria coinfection and intestinal inflammation to cecal colonization and systemic spread of salmonella typhimurium deficient in tetrathionate reductase or type III secretion systems salmonella pathogenicity island 1 or 2

Claire-Sophie Rimet, John J. Maurer, Roy D. Berghaus, Brian J. Jordan, Luciana Helena Antoniassi da Silva, Lisa J. Stabler, Kasey K. Johnson, Laura R. Tensa, Karen M. Segovia, Monique S. Franca


										

The contribution of eimeria coinfection and intestinal inflammation to cecal colonization and systemic spread of salmonella typhimurium deficient in tetrathionate reductase or type III secretion systems salmonella pathogenicity island 1 or 2

Claire-Sophie Rimet, John J. Maurer, Roy D. Berghaus, Brian J. Jordan, Luciana Helena Antoniassi da Silva, Lisa J. Stabler, Kasey K. Johnson, Laura R. Tensa, Karen M. Segovia, Monique S. Franca

    Fontes

    Avian diseases

    Vol. 63, no. 4 (June, 2019), p. 559-567