Urinary dysfunction in transgenic sickle cell mice : model of idiopathic overactive bladder syndrome

Urinary dysfunction in transgenic sickle cell mice : model of idiopathic overactive bladder syndrome

Serkan Karakus, Uzoma A. Anele, Fabio H. Silva, Biljana Musicki, Arthur L. Burnett

ARTIGO

Inglês

Agradecimentos: This work was supported by National Institute of Diabetes and Digestive and Kidney Diseases Grant R56 DK-114095 (to A. L. Burnett) and São Paulo Research Foundation Brazil Grants 2017/08122-9 and 2018/06243-6 (to F. H. Silva)

Voiding abnormalities are common among the sickle cell disease (SCD) population, among which overactive bladder (OAB) syndrome is observed at rates as high as 39%. Although detrusor overactivity is the most common cause of OAB, its molecular pathophysiology is not well elucidated. The nitric oxide...

FUNDAÇÃO DE AMPARO À PESQUISA DO ESTADO DE SÃO PAULO - FAPESP

2017/08122-9; 2018/06243-6

Fechado

Urinary dysfunction in transgenic sickle cell mice : model of idiopathic overactive bladder syndrome

Serkan Karakus, Uzoma A. Anele, Fabio H. Silva, Biljana Musicki, Arthur L. Burnett

										

Urinary dysfunction in transgenic sickle cell mice : model of idiopathic overactive bladder syndrome

Serkan Karakus, Uzoma A. Anele, Fabio H. Silva, Biljana Musicki, Arthur L. Burnett

    Fontes

    American journal of physiology: renal physiology

    Vol. 317, no. 3 (Sept., 2019), p. F540-F546